MEDICAL MALPRACTICE 5
The physician should have expressly required closer monitoring of vital signs, presence
of ocular palsy and papilloedema, serum oxygen saturation, urinary output, and level of
consciousness (LOC) and neurological status (q. 2 hours). The latter includes assessment of
reflexive patency, miosis, vertigo, confusion, slurred speech and Cushing’s triad, all of which are
proxies of intracranial pressure (ICP) (Logan, Bell & Leonard, 2001; Czosnyka & Pickard,
2004). Furthermore, the ED physician should have administered 80 g. Mannitol to protect against
an ICP surge, performed radiographic tests and referred the patient to a supervising neurologist
for specialist evaluation.
The Case for the Plaintiff: Arguments and Evidence
In arguing the case for the plaintiff’s family, I would posit that the care provided to him
was substandard and was the proximate cause of his death. Although the patient was alert at the
time of presentation, he was intoxicated and had just been rescued from a traumatic car accident.
If the physician had cared to perform toxicology screening (urine and serum tests), s/he would
have established that the patient had also ingested chlordiazepoxide (Librium) and would have
been more cautious in the administration of methadone, despite the intoxicated patient’s
protestations. Indeed, while it can be argued that the patient misled the physician regarding his
drug withdrawal treatment program, the physician is legally required to confirm drug history
from program records. Besides, as the patient was both a delinquent and engaged in underage
alcohol drinking, the practitioner should have practiced extra caution in taking the teenage
patient’s word as truth.
As the plaintiff’s legal representative, I would call on experts and respected practicing
physicians to demonstrate the typically accepted standard of care in this scenario. Depositions
would include how a pertinent history would have influenced medication, the legal obligation of